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Chapter 1 advent (pages 1–3): W.F. Bodmer
Chapter 2 Expression of Embryonic Characters via Malignant Cells (pages 4–27): Francois Jacob
Chapter three Embryonic Antigens in Malignancy and being pregnant: universal Denominators in Immune law (pages 28–54): Joseph H. Coggin
Chapter four The T/t?Complex: a kinfolk of Genes Controlling Early Embryonic floor Antigens (pages 55–68): Karen Artzt
Chapter five Fetal his Tocompatibility Antigens and Maternal Immune Responses (pages 69–88): W.D. Billington and S.C. Bell
Chapter 6 How are Tumour Antigens regarding general Antigens? (pages 89–103): E.S. Lennox
Chapter 7 The sensible importance of Tumour?Associated mobile floor changes of Embryonic and Unknown beginning (pages 104–123): Reinhard Kurth
Chapter eight Host popularity of Fetal Antigens: Do they set off particular Antibodies? (pages 125–144): L. Brent, R. Hunt, I. V. Hutchinson, P. B. Medawar, L. Palmer and L. Welsh
Chapter nine Human Chorionic Gonadotropin as a version for a Fetal Antigen (pages 146–159): ok. D. Bagshawe
Chapter 10 Can Fetal Antigens be Used for Prophylactic Immunization? (pages 160–181): P. B. Medawar and Ruth Hunt
Chapter eleven Parity and Susceptibility to melanoma (pages 182–203): Valerie Beral
Chapter 12 Human Antigens well-known via Monoclonal Antibodies (pages 204–229): Hilary Koprowski
Chapter thirteen Antibodies to Fetal Antigens linked to Rodent Tumours (pages 230–247): R. W. Baldwin
Chapter 14 last comments (pages 248–252): W. F. Bodmer

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Extra info for Ciba Foundation Symposium 96 - Fetal Antigens and Cancer

Sample text

Goodfellow: The cellular transforming genes (normal homologues of the viral oncogenes) may not form a homogeneous group. Cooper (1982) has recently reviewed their expression in normal and tumour cells. Kurth: Sometimes several onc genes are expressed in a single tumour, and nobody knows which, if any, of the onc gene products contribute to the resulting phenotype. It is too early to discuss this in detail; what is needed is for one group to study all of the 15 known onc genes and use all these probes for individual tumours of defined histopathology.

In hamsters and mice the protective Tc lymphocyte population was removed by treatment with a n t i 4 antiserum plus complement and these cells carried the Lyt 2+3+ phenotype in mice (Coggin et a1 1980). From these combined results there is no doubt that EAs expressed on fetal cells in utero serve to sensitize Tc lymphocytes in pregnant females and thus function as potent T-cell immunogens. That such determinants can successfully pass the placental barrier is no longer in doubt. There seem to be a number of EAs expressed on the tissues of the fetus.

As I understood it, you said that some of these responses cannot be found in the first pregnancy, and you didn’t comment on the transplacental transfer of the antibody. Coggin: We have not done the appropriate studies with the antibody to answer this question adequately.

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